Modifiers of C9orf72 dipeptide repeat toxicity connect nucleocytoplasmic transport defects to FTD/ALS
Stanford University · Salk Institute for Biological Studies · +5 more institutions
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Abstract
No abstract available for this paper.
Citation impact
636
total citations
- FWCI
- 54.05
- Percentile
- 100%
- References
- 27
Citations per year
Authors
15- AJAna JovičićCorresponding
Stanford University
- JMJérôme Mertens
Salk Institute for Biological Studies, Sanford Consortium for Regenerative Medicine
- SBSteven Boeynaems
KU Leuven, Vlaams Instituut voor Biotechnologie, VIB-KU Leuven Center for Brain & Disease Research
- EBElke Bogaert
Vlaams Instituut voor Biotechnologie, VIB-KU Leuven Center for Brain & Disease Research, KU Leuven
- NCNoori Chai
Stanford University
Topics & keywords
Topics
Keywords
- C9orf72
- Neurodegeneration
- Frontotemporal dementia
- Amyotrophic lateral sclerosis
- Trinucleotide repeat expansion
- Neuroscience
- Biology
- Translation (biology)
UN Sustainable Development Goals
- Good health and well-being
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