Rapamycin induces feedback activation of Akt signaling through an IGF-1R-dependent mechanism
National Institutes of Health · Center for Cancer Research · +1 more institution
Indexed incrossrefpubmed
Abstract
No abstract available for this paper.
Citation impact
747
total citations
- FWCI
- 13.36
- Percentile
- 100%
- References
- 39
Citations per year
Authors
5- XWXiaolin WanCorresponding
National Institutes of Health, Center for Cancer Research, Molecular Oncology (United States)
- BHB Harkavy
National Institutes of Health, Center for Cancer Research, Molecular Oncology (United States)
- NSN Shen
National Institutes of Health, Center for Cancer Research, Molecular Oncology (United States)
- PJPatrick J. Grohar
National Institutes of Health, Center for Cancer Research, Molecular Oncology (United States)
- LJLee J. Helman
National Institutes of Health, Center for Cancer Research, Molecular Oncology (United States)
Topics & keywords
Topics
Keywords
- PI3K/AKT/mTOR pathway
- Protein kinase B
- Biology
- Phosphorylation
- P70-S6 Kinase 1
- Small interfering RNA
- RPTOR
- mTORC1
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