Aβ42‐driven cerebral amyloidosis in transgenic mice reveals early and robust pathology
Hertie Institute for Clinical Brain Research · University of Basel · +7 more institutions
Indexed incrossrefdoajpubmed
Abstract
No abstract available for this paper.
Citation impact
1,007
total citations
- FWCI
- 7.49
- Percentile
- 100%
- References
- 32
Citations per year
Authors
16- RRRebecca RaddeCorresponding
Hertie Institute for Clinical Brain Research
- TBTristan Bolmont
Hertie Institute for Clinical Brain Research
- SAStephan A. Kaeser
Hertie Institute for Clinical Brain Research
- JCJanaky Coomaraswamy
Hertie Institute for Clinical Brain Research
- DLDennis Lindau
Hertie Institute for Clinical Brain Research
Topics & keywords
Topics
Keywords
- Gliosis
- Amyloidosis
- Genetically modified mouse
- Presenilin
- Amyloid (mycology)
- Transgene
- Senile plaques
- Pathology
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