Keap1 loss promotes Kras-driven lung cancer and results in dependence on glutaminolysis
Massachusetts Institute of Technology · New York University · +10 more institutions
Indexed incrossrefpubmed
Abstract
No abstract available for this paper.
Citation impact
660
total citations
- FWCI
- 25.45
- Percentile
- 100%
- References
- 71
Citations per year
Authors
28Topics & keywords
Topics
Keywords
- KRAS
- KEAP1
- Carcinogenesis
- Cancer research
- Regulator
- Lung cancer
- Biology
- Glutaminolysis
UN Sustainable Development Goals
- Good health and well-being
No related works found for this paper.
Funding
- NSNational Science FoundationAwards: 1122374, P30-CA14051
- HFHope Funds for Cancer Research
- WSWenner-Gren Stiftelserna
- KIKoch Institute for Integrative Cancer Research, Massachusetts Institute of TechnologyAward: P30-CA14051
- ECEuropean CommissionAwards: GA-2013-609409, LTCOFUND2013, 609409
- SAStiftelsen Assar Gabrielssons Fond
- NINational Institutes of HealthAwards: 5T32HL007151-38, K22CA201088, P30-CA14051
- MRMedical Research Council
- NCNational Cancer InstituteAwards: P30-CA14051, CA14051