mTOR controls mitochondrial oxidative function through a YY1–PGC-1α transcriptional complex
Dana-Farber Cancer Institute · Harvard University · +5 more institutions
Indexed incrossrefpubmed
Abstract
No abstract available for this paper.
Citation impact
1,490
total citations
- FWCI
- 18.57
- Percentile
- 100%
- References
- 33
Citations per year
Authors
6- JTJohn T. CunninghamCorresponding
Dana-Farber Cancer Institute, Harvard University
- JTJoseph T. Rodgers
Harvard University, Dana-Farber Cancer Institute
- DHDaniel H. Arlow
Massachusetts Institute of Technology, Harvard University, Broad Institute, Massachusetts General Hospital
- FVFrancisca Vázquez
Harvard University, Dana-Farber Cancer Institute
- VKVamsi K. Mootha
Massachusetts Institute of Technology, Broad Institute, Massachusetts General Hospital, Harvard University
Topics & keywords
Topics
Keywords
- PI3K/AKT/mTOR pathway
- PPARGC1A
- Biology
- NRF1
- Oxidative phosphorylation
- Cell biology
- RPTOR
- Mechanistic target of rapamycin
UN Sustainable Development Goals
- Affordable and clean energy
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