articleScienceNov 24, 2005Closed access

The Kinase LKB1 Mediates Glucose Homeostasis in Liver and Therapeutic Effects of Metformin

Salk Institute for Biological Studies · Beth Israel Deaconess Medical Center · +6 more institutions

PubMed
Indexed incrossrefpubmed

Abstract

The Peutz-Jegher syndrome tumor-suppressor gene encodes a protein-threonine kinase, LKB1, which phosphorylates and activates AMPK [adenosine monophosphate (AMP)-activated protein kinase]. The deletion of LKB1 in the liver of adult mice resulted in a nearly complete loss of AMPK activity. Loss of LKB1 function resulted in hyperglycemia with increased gluconeogenic and lipogenic gene expression. In LKB1-deficient livers, TORC2, a transcriptional coactivator of CREB (cAMP response element-binding protein), was dephosphorylated and entered the nucleus, driving the expression of peroxisome proliferator-activated receptor-gamma coactivator 1alpha (PGC-1alpha), which in turn drives gluconeogenesis. Adenoviral small…

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1,932
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Authors

8

Topics & keywords

Keywords
  • AMPK
  • Gluconeogenesis
  • Endocrinology
  • AMP-activated protein kinase
  • Glucose homeostasis
  • Internal medicine
  • Protein kinase A
  • Coactivator
UN Sustainable Development Goals
  • Good health and well-being
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