reviewJournal of Cellular PhysiologyFeb 20, 2016Closed access

Mitochondria and Mitochondrial ROS in Cancer: Novel Targets for Anticancer Therapy

Union Hospital · Heidelberg University · +5 more institutions

PubMed
Indexed incrossrefpubmed

Abstract

Mitochondria are indispensable for energy metabolism, apoptosis regulation, and cell signaling. Mitochondria in malignant cells differ structurally and functionally from those in normal cells and participate actively in metabolic reprogramming. Mitochondria in cancer cells are characterized by reactive oxygen species (ROS) overproduction, which promotes cancer development by inducing genomic instability, modifying gene expression, and participating in signaling pathways. Mitochondrial and nuclear DNA mutations caused by oxidative damage that impair the oxidative phosphorylation process will result in further mitochondrial ROS production, completing the "vicious cycle" between mitochondria, ROS, genomic…

Citation impact

685
total citations
FWCI
17.93
Percentile
100%
References
148
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Authors

7

Topics & keywords

Keywords
  • Mitochondrion
  • Oxidative phosphorylation
  • Cancer cell
  • Cell biology
  • Mitochondrial ROS
  • Biology
  • Reactive oxygen species
  • Apoptosis
UN Sustainable Development Goals
  • Good health and well-being
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